Healing the Sick of the Sick: Inside Dr. Crozier’s Approach

You know, I thought we were supposed to be happy for our people and our patients when they got better, no matter if I get them better or somebody else. Yeah, better. You know, I'm always excited when somebody gets better, you know, and I don't have to be the one that gets them better for me.

Yeah, if you can learn something from someone else, awesome.

So we got to learn from each other, and I don't understand the hostility. Instability between all the physicians, you know, and why we're trying to cut each other.

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Hello, this is Dr. Kent Holtorp with another episode of the Peptide Summit. Today we welcome Dr. Gordon Crozier. He's a big deal. We've had a lot of great people that have knowledge, but he's a leading medical practitioner, researcher, and lecturer in areas of integrative and functional medicine as it relates to cellular therapy. And people come from all over the world to see him. His knowledge base is truly incredible. His lectures just blow people away. So we're very happy to have you. Thanks. Thanks for being on. I'll finish your bio. But really appreciate you taking the time.

Thank you. It's good to be here.

Great, great. He looks great too, you know, at his practice. I think he treats like the sick of the sick kind of, you know, bring it on type clinic in Orlando, Florida. He's a pioneer in the research and application of peptide therapy, revolutionary therapy that stimulates cellular regrowth in systems. He's recognized as the country's leading authority in emerging treatments, is highly sought after nationally and internationally. And I know he's a highly sought-after speaker. He's a bestselling author. He's been featured on national TV, radio, journals, and medical publications. So we're so happy to have him. And he's gonna talk today about Inside Secrets Revealed with Actual Patient Case Studies. So we're gonna hear basically about, you know, true studies and, and what he did to get these patients better. And I imagine you're not the first stop for anyone, that they've been to many doctors.

So yeah, most of my clientele, most of my patients have been around the block. They've been to all the big name hospitals and research centers and really had minimal results to no results. And some of them have been to 22+ physicians in the past, and including some of them integrative medicines in the past. And they just can't, they can't get well. So I like the challenge, I guess, because, you know, it's all about cellular health, as you were saying. It's all about the cell and how to repair the cell so you can help and heal the whole organ systems. So that's, that's, I like to kind of find what's actually triggering what.

And it sounds like you're really You know, like the House type, they'll, uh, doctor, a medical detective. And I know it really takes passion and, you know, you're tired, you go, oh, I'm sure you're hitting the research. I gotta find out what this, what, how I can help this woman.

Yeah.

I think it's awesome. And that's what, you know, being a doctor is, right? That's not what it is now. Being a doctor now is algorithm and, you know, here you go. But I just love the fact that, that you do that and you've been doing it a a long time and just a great knowledge and so generous in sharing all your knowledge. So again, again, thank you. Do you have any favorite peptides?

Oh, wow.

I don't know.

You use a lot of peptides. I use a lot of peptides, a lot.

It's kind of your favorite.

Really, my favorite— probably my favorite of all time is thymus and Alpha-1. And I use it so routinely on myself and so many different patients. And I love what it does. And especially right now when we're worried about our immune systems and all that, really helps to to modulate that immune system so it won't overdrive it. And that's—

Can you talk a little more about the mechanism, like how it works and, or compared to the other thymosins or—

So thymosin alpha-1 actually is a modulator and it helps to balance the Th1, Th2 pathways for, because you need some inflammation in your immune system, but you don't want it overdriven so that you actually will drive it into an autoimmune response. So that's why thymosin alpha-1 will work a lot of times with people with autoimmune diseases, which I use a ton of it in autoimmune diseases because it helps dampen down that overdriven immune system. So it, you know, it actually helps with every aspect of the immune systems and your natural killer cells. It'll modulate them and it modulates them. It doesn't overdrive the natural killer cells. It just modulates them so that they will work correctly. And so I love it because it works so perfectly and I've used it, you know, they've actually been out of it several times recently because I think everybody's using it now. And actually that's really helped a lot with my cancer patients because I get referrals from some of the cancer centers here in Florida for people that are undergoing chemotherapy that these doctors know that they're exposed. And so I add thymosin alpha 1 into that, and they don't end up with the pneumonias and other diseases because of their chemotherapy.

I'll make a disclaimer: we're not saying it treats cancer.

No, it doesn't treat cancer.

Yeah, we're, you know, but it's approved for cancer in other countries, but we're not saying it treats it. But it's interesting, when you look at, uh, cancer relapse, it's basically the level of your natural killer cell function is going to determine your relapse level. So chemo destroys your immunity.

Yeah.

And then they do nothing about it. And the nice thing is, is, yeah, that this will boost it. But, you know, people think of— or they go, I got autoimmune, I don't want to boost my immunity. I think it's kind of like all or one, but it's like a teeter-totter, you know?

Right.

And so boost the good immunity and lowers that inflammation and that autoimmunity. And, and yeah, so that's, that, that's great. I've been around tons of studies on it.

Yeah, there's tons of studies on it. It's been studied in over 70 countries. And that's why the cancer doctors around where I live here, even from the other coast, you know, kind of refer patients because they do.

I mean, I— yeah, an oncologist, like you try to give them anything and they go, don't touch my patient.

You know, well, I'm not going to without their approval and I never do. We always talk beforehand.

Don't blame it, you know?

Yeah. So they always talk to me beforehand. They'd say, I'm sending so-and-so patient. They had a stem cell transplant for whatever disease, like leukemia, multiple myeloma, something like that. They said, we know you can help with some of these things and boost them and help them. And we would like you to do that. Just let me know what you think. And then I would call them back after I evaluate them and I said, I think that these modalities, including thymosin alpha 1, would be good. A lot of them never heard of thymosin alpha 1 before.

That's what I'm saying. I'm like, am I dreaming? They're actually— oncologists will call and say, oh, can you see my patient? You say no. And you can send, like, at least my experience is they never want to read a study. They don't care. They look at the algorithm. And it's weird to me is that they don't even test like what chemo or whatever is the cancer most sensitive to. They just give their, their recipe, you know.

Yeah, their recipe.

And then if, you know, I'll give the patient here, here's some studies and they, no, he wouldn't read them, you know, just don't do it.

And so, well, they won't read them. I'll give them to them, but they just want me to give them the gist of what they say. So, you know, it's up to us to read.

Step That's a step forward.

Yeah, that's good. So yeah, what are the ones do you like?

What are the peptides you use a lot of?

Well, I use a ton of BPC-157. I use it both orally and injection because I see athletes too, because I lift weights. So I'm in the gym a lot. And so I have all the gym rats.

What is that, a gym? I've heard of that.

The gym rats.

I think, I think I saw one a couple of years ago.

So we, you know, you know, I just, I like to work out. So that's kind of one of my things that I release. And so I use a lot of BPC-157, you know, for a lot of these people that strain muscles, strain tendons, you know, it really helps them to recover from a lot of those things. And I love it because it works for that.

I think it's a double-edged sword because, you know, and all these bodybuilders and things, they're way ahead of the curve. In terms of their knowledge versus doctors. Like, they know so much and they use it. So it's like, unfortunately, it can get thrown in with, oh, it's for bodybuilders, you know, that L thing, you know, and it just helps recovery, you know, you know, but also boosts mitochondrial function, other things too. But yeah, it's like they find out what works and then everyone goes, oh, it's a bodybuilder thing, you know. Or that's what, like, someone's doctor will say.

Yeah, that's what somebody else will say, right?

Yeah. Yeah.

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But that's, that's awesome. And, and the kind of cases you see, everything from neurologic to hormonal. Uh, it's all Lyme, Sears.

Uh, obviously there's probably 80% of my practice Lyme and Sears. And, um, because every single Lyme and Sears patient, they all end up with some type of autoimmune disease. So they all have some autoimmunity.

Oh man, I just love— because I've given lectures on that and they're basically all shown to have, you know, antibodies to the brain, right? Pituitary antibodies, hypothalamic antibodies. But, you know, you need either a special test, like one's the brain, or there is no test for the ones like to the hypothalamus and pituitary.

Yeah, research.

But yeah, and they'll get, you know, they'll go to the rheumatologist and get a diagnosis because they'll have a bunch of different antibodies to the autoantibodies and that where they come and go and they'll get diagnosed with, you know, mixed connective tissue disease, meaning, well, it's autoimmune, but we don't know what it is or antiphospholipid syndrome. They say you're gonna have it for life. Well, I'm like, no, you won't. No. Yeah. And it is, they have these titers that just go up, up and down. And it goes along with it. And, and we've kind of changed. It sounds like you have too. Now, I started because I had Lyme, and I sure want to know why you kind of got into this. But, you know, I did antibiotics, just massive antibiotics, and it didn't work because my natural killer cell function was like 1.

Right?

And there's no immunity to kill it. So you can never— doesn't matter how much, you know, antibiotics you give. But we've become really an immune modulatory cells. And it works for— it's like all the labs you do that on, you know, chronic fatigue syndrome, fibromyalgia, Lyme, Alzheimer's, autism. And yeah, damn, everyone's immune system is thrashed. You know, toxins, the seer. Oh, it's just It's bad. But yeah, kind of going backward, but interesting. What kind of got you into this crazy medicine?

Well, it was kind of really my own story and what happened to me. And I didn't even realize it until I was clinical faculty at the University of Kentucky and I got sick. But I, you know, I had always been— had undulating health ever since I was in the second grade. In the second grade, this is back before anybody knew about Lyme disease. I was bit by a tick in Minnesota, which is one of the hotbeds, and I suddenly couldn't read. So I had, I had to be taught how to relearn how to read. I was, went from the top of my class to the bottom, being called a retard by everybody in my class, because you could actually do that back then.

And how old were you?

I was in second grade. So I was, uh, boy, about 7, 8 years old.

And they of course say, okay, why would you all of a sudden regress?

Right. Why would you regress? Well, nobody's tried to figure it out. Nobody's like, yeah, okay.

Yeah. Go to rehab, whatever. Yeah.

So I always had undulating health all my life. So I was clinical faculty. I was actually setting up rural women's health clinics throughout Tennessee, Kentucky, uh, West Virginia and Southern Ohio. And trying to help with rural clinical help to help people in these areas with their health. And it was difficult. It wasn't an easy, easy go. But so that, that was my job primarily there at the university. And then we taught residents and all that stuff, but I got, I got really sick. I was doing, I was doing surgeries and I had some residents with me and I was teaching them some laparoscopic techniques, and we were starting to do robotics back then.

So you were a surgery resident?

Yeah, I was OB-GYN.

Oh, OB-GYN.

So I did gynecological surgeries using the robots and, you know, robotic surgeries. And so I taught the residents and I started getting so much pain and I just kept moving forward, you know, yeah, I'm in a lot of pain, unbelievable pain. And I noticed I was kind of forgetting some things a little bit at that time. But what happened was I just started passing out for no reason at all. And I passed out a couple of times in surgery. I went on medical disability, of course.

Yeah, it doesn't look good. That's not— yeah, you don't want your surgeon doing that.

Yeah. Yeah. Why are you a surgeon passing out? So I went on medical— nobody could figure it out.

It is arrhythmia or what?

I had no arrhythmias that they could find. I think it was really POTS. And I think the thing was, is that nobody really figured it out.

And then no one tries to figure it out as a problem.

So, you know, so I began doing my own testing. So this is back in 2007, 2007, 2006, 2007. And so I started doing my own testing on myself and I'm going like, oh my goodness, I have, I have Lyme, Lyme disease. And oh my goodness, my inflammatory markers are really out of control. And so I started looking at my inflammatory markers and I'm going like, wow, I've got something else, you know, pushing down my immune system. I don't have an immune system. And so, you know, I kind of figured it out on my own. And I, you know, I kind of listened to some other— there wasn't a lot of Lyme doctors back then. There were a few, but, you know, just trying to figure it out.

But when would you also say that You know, I, I really think that, you know, people say, oh, what specialty? It doesn't matter to you, but it matters what you learn after you're out.

Yes.

And it's, it's just incredible. I really think you can look at old literature and be a better specialist in about 2 months than most specialties, you know, not surgery or something like that. But, you know, all these other ones, because they're just stuck You know, doing what they were taught in residency.

Yeah.

And the only studies of— they only read the studies they say they do is a drug rep comes in, you know, it's something new on the formulary. And I did have a, you know, basically a paper on it. You know, what doctors know, why? Because people go, why doesn't my doctor know this? And in Annals of Internal Medicine, look, they found that doctors are practicing 20 years behind what's available in medical literature. They do not read medical literature even though they say they do. And when they do, they may read the abstract, but they really don't. But even worse is that if you give a doctor a— here's 10 studies showing what you're doing is not the best treatment— they don't want to hear it. No, no, no. My patients are different. And, you know, it's even got into the L.A. Times. They, you know, the doctors that don't want to follow— they say they're evidence-based. But you give me evidence and they go, no, I don't believe that one because it's not what I do. I don't believe that one.

Right.

And which is why you look at levels of evidence, you know, basically have to randomize double-blind placebo-controlled. Then you got meta-analysis and single-blind and case studies. And then you have societal recommendations.

Yeah.

Which are the worst level of evidence. And what do most doctors go by? Oh, our, you know, Cardiology Society, Endocrine Society says do this. It's the worst level because they don't change their opinion based on new evidence. They're basically— they, they have a position and they don't change no matter what comes out. It's frustrating. But you sound like you've got a good mix, good relationship with the standard docs.

I try because, you know, we have to, you know, I've gone through it because I actually was reported to my board by a plastic surgeon who didn't believe that the breast implants— that the woman I was seeing, she had breast implants. And I'm going, I've done everything for you. I swear it's your breast implants. I just go back to them and have them, you know, see if they are, you know, it's your ask. She went back and asked him, can my breast implants be making me sick? And he reported me to the board saying everything I was doing was a bunch of hoax and da da da da. So, you know, we all have to go through those fights. So because of that, I'm trying to make friends with my colleagues around here to let them know that this is a valid form of medicine. All I'm doing is reading research and seeing what can be applied to the human person so that they can live better, a better life. I don't say I can cure anybody. I don't say that I can make them totally well or heal them, but I can tell you I can make you have a better life.

I like that. I like that.

So let's talk a little bit about Parkinson's. You know, it's a lot of people go on a lot of Parkinson's meds, and this was a 74-year-old who was really on a lot of meds and he'd been on them for a long time. Well, you know, the longer they're on them, they really don't work very well. And that was his big complaint and why he came to me. So he came to me because, you know, his meds really were not working well for him. He would take them and 2 hours later he'd start to get rigidity and cogwheeling and he couldn't do it, but he was having problems focusing. He was always getting a little bit of depression too. He was a very analytical guy, so he would put everything out and have everything very strategic. Every time he came to me, you know, it says at this time, at this time, you know, he had everything in 15-minute increments all day long for weeks on end. And, you know, he expect me to look at all these graphs and I'm like, oh my. A little bit over the top. But, you know, he was a smart guy, very smart. He built a cockpit in his basement and all these things. But, you know, so he came in, he really wanted help.

Because I just think what he feels like, he just was a brilliant guy. And now he's just like, frail, debilitated, can't write. But yeah, and they just give meds and go, go away.

Yeah, here's your meds, go away, you know. And he had been to a lot of places. And I won't say some of the hospitals that I went to.

Oh, yeah, they go, I'm going to the Mayo Clinic. All right.

I'll see you tomorrow. Yeah. So, you know, he came in, he did have some elevated inflammatory markers. You know, his TGIF beta was elevated. He had inflammation. His C4a was elevated. He had— so, you know, you were thinking, oh, does he have Sears? What does he have?

Yeah. And I just want to mention those are inflammatory markers that most doctors will say, oh, inflammatory marker, C-reactive protein, and that's all they check, which we find isn't often elevated in these patients. But so these are different, more specific inflammatory markers, right?

Right. So they're inflammatory markers that I look at. I, I look at all the inflammatory markers and, you know, everything's inflammation.

You look at all the diseases, exactly.

And so that's what I find out with a lot of the Parkinson's patients is they have a lot of neuroinflammation. So they have neuroinflammation. So naturally when you have neuroinflammation, nothing is really going to work quite correctly. So he had a lot of it. And in fact, his C4a was 1,960. I'm just looking at it right here. I pulled it up.

And what lab?

This is through Quest. Yeah, that's Quest. So the top edge—

you have problem getting that?

Now I am. This is, this is a year and a half ago.

So it did like all the key tests. It's like, you know, natural killer cell function. Yes. EFA. Human growth factor beta. Those are the three they screw up every time.

They always mess them up. Always mess them up. And sometimes if I get like, when I got this back, this was a repeat because I said, oh, that's way too high. They didn't do something right. Let's do it again. So I said, let's draw it in our office 'cause I can do that and they come and pick 'em up so that I can actually know that this is really, it was processed correctly.

Yeah, those have to be processed correctly, yeah.

So we did it and it actually was the same. So I guess it was right.

Or just finding it. I went to Quest myself and the phlebotomist was, oh, this is that doctor that writes for so many tests. Yeah, yeah. I'm like, oh yeah, and I heard he's really cute. But, and then, and then she goes, oh, let me send off your nitric oxide killer cell, I'll be right back. Like, okay, that's great. Came back like no specimen received, you know. Right. Anyway, yeah, that's a request, but they're all Terrible.

Yeah, it doesn't matter what lab you use anymore. It's just, you know, it's independent. So, so he really, he was just desperate for something. His C-reactive protein was actually elevated too. It was all up, it was 11.4. So, you know, he had a high C-reactive protein. His homocysteine was 14. I'm just looking at his labs right now. I pulled them up. And so he had high inflammation even on inflammatory markers that normal people look at, you know, the normal physicians that don't look a little bit deeper. So, you know, he just really wanted help. He wanted something. And, you know, they've done lots of studies on glutathione and glutathione and how it works. And we've all probably seen some Parkinson's patients. If you do an IV glutathione, they're immediately, they're pretty good immediately, but it's only going to last a very, very short period of time. So it's really not, you know, it's not good, you know, because we need something that's going to work a little bit better.

Yeah. And those IV studies were out a long time ago.

A long time ago.

Yeah, it's again, it's that old research.

Yeah, it's old research.

And if— have you found giving NAC continuously to Parkinson's patients is beneficial, which converts to glutathione?

Yeah, well, I will do that because most people— the NAC, it's the cysteine component. So a lot of people, when you study most people, because I always do an amino acid profile on everybody, and you might actually like So, you know, I'm going to get into the tests you like later, just so you know. Oh, okay. Why? You love the ones.

I love to hear it.

Yeah, I try to use ones where their insurance is going to pay for it. So I will use amino acid profiles from Quest and LabCorp.

Oh, just, just through standard.

So I'm doing for the standard ones if I have to, but then I also use something called Vibrant of America. It's probably, it's kind of probably one of my favorites.

So I've heard of America.

Yeah. So I use a lot of their testing for their environmental testing too. I always do amino acid profiles because if I'm going to be giving any peptides, peptides are amino acids in chains. So if you are short on amino acids, a lot of times those peptides are going to break up to provide the amino acid is actually needed in the body. And then you don't really get the full benefit, the full benefit. You might get some, but you're not going to get the full benefit of that peptide. So that's why I always do that so I can actually optimize their full amino acid profile first because I think it's good for us to know, and I can't necessarily remember every time, oh yeah, that is a 15-chain peptide and it has, you know, leucine, Or like, no, I can't remember those. So I do look them up from time to time to make sure that that person, if that person is deficient on amino acids, I want to make sure it's not one of the ones that I'm going to be giving in a peptide.

So is your thought that it will take it from the peptide?

Yes.

And do you replace with amino acids IV or orally?

Yes, I do. I will do it first IV-wise and then do it orally.

So, so I think that helps with, with everything. And I think we, when we were talking before, how, you know, we were talking about how the peptides are really synergistic with so many different therapies. And while, while they'll work on their own, but they, they basically, it's exponentially with, with every other treatment.

I think so. I think exactly that way.

Yeah, and I mentioned a doctor came to where one of the conferences and said, talked about BPC-157 and said, oh my gosh, it makes everything work better, you know. And I'd never met her, it was Dr. Tringali who, uh, uh, we're going to interview, uh, next. But, uh, yeah, she's like just in love with it. It just— yeah, yeah, it makes— it works great on its own, but even better when you use it with other therapies.

Yeah. Exactly. And you can take that orally. It's very stabilized orally. I mean, most of them, you're getting them stabilized to take orally. And that's what I love about what you do is because you are actually going to the next level to actually help these things be stabilized for oral use. And that's what's so amazing about you.

So I think I could have retired, but the money we're spending. Yeah, but no, I love it too. It's kind of like, you know, researching and trying to make, you know, better mousetrap. Yeah.

Yeah. Yeah. So the, you know, because he had such high inflammation, of course, you know, I put him on oral regimens and I put him on oral, he was on NAC, of course, but I also really did some IV therapies with him too of amino acids and, uh, some other things so that we could actually—

Like ozone?

I do like ozone.

Yeah, that's one of our main things. I think it goes along well with everything.

I think ozone works well with most treatment modalities, and I think it really augments a lot of them. So I love ozone. But I started him actually on thymus and alpha-1. It was the first one I started him on. And I started him on that first because of his inflammatory markers. I wanted to make sure we were modulating things. And just looking at, you know, even his monocytes, monocytes I look at for gut and bowel inflammation because there's some evidence. You probably heard Dr. Lavelle talk about it many times, Jim Lavelle, that if your monocytes are over 7, you probably have some bowel inflammation. And so, you know, and I look at that, I look at that on every single person because there's a lot of people, they don't have bowel issues, but they might have a little bit of inflammation there. That little bit of inflammation in their bowel is going to help them is going to make them not create some of the neuropeptides that they neuro— they're all so connected. It's so connected.

I mean, I even stopped doing like, you know, food sensitivity because they're positive on everything. Everything, all these big proteins coming through. It's like on everyone.

Yes.

Oh, even pretty healthy people. You know, it's all this. you know, American diet and stress and whatever else, toxins, pesticides, everything.

Right.

But everyone's bowel's messed up and I think it's a big part of the whole vicious cycle.

Right, right. So I started him on that thymosin alpha 1, but then I also put him on C-Lanc and C-Max.

Can you talk about those?

I love C-Lanc and C-Max. So C-Lanc, it is a modulator. There's some evidence, you know, I just I have an article that actually I pulled up a couple weeks ago and it actually was, it was on how C-Lanc affects GABA, but C-Lanc also affects genetic expression for neurotransmitters. And I had not seen this article before and it was way back from 2018. And I'm going like, why did I not see this article before?

Well, there's so many. I mean, just, wow. No. Yeah.

That's why I try to read at least 6 to 8 every morning. You know, that's my morning routine. You know, I'm going to read 6 to 8 articles.

You do every morning more than most doctors do in a year.

Yeah.

Just to let you know. I mean, that's totally true.

But you're falling behind if you don't.

You—

that's why I think I don't want to— I still feel like I'm falling behind. And that's why, you know, you know, putting things by you, you, you find different articles than I do. And so I have, you know, different people that can say, hey, have you seen this article? This is a great article about, you know, what—

I love that when people send me or patients find something because I remember 15, 20 years ago, I remember it used to be like whoever— you have to go to the library, find the journal, which often wasn't there, take it and stick it on the copy machine. Now it's information overload, right? There's so much you have to keep looking at all these services that, you know, it's just, that's the problem of so many stuff thrown at me. It's like trying to take the time to go just sort through them even to see which ones you want to read. And yeah, if I were to pan over the stacks of journals that are my to-read pile, it's, it gets crazy.

I'm sure it does. It gets very crazy. And then trying to find out, you know, then you spend this time reading this article and you found out you, you you figure it out, oh my goodness, it's not even statistically significant. I spent all that time on that article and it's no good. Yeah.

Yeah. So, part of the gig.

So I like C-Lanc because of that. And it also helps to increase bone-derived neurotrophic factors. So, you know, it's the—

between C-Lanc and C-Max, they're both good for brain. I mean, I noticed that with C-Lanc, like when I'm stressed, I'm like, where's my C-Lanc? You know? Right. Yeah. And it was—

there's a lot of— yeah, there's a lot of evidence that it's an anti-anxiolytic. So, you know, and I will use it on my patients that are overly anxious too. And it's very easy to use, just, you know, nasal spray, you know, just carry it around with you, put it in a nasal spray, but they say it works after it's out of the fridge. I don't know. Yeah.

And I don't think I've ever seen a side effect from them. No. Yeah.

No, I haven't.

Yeah. Injection. We're working on oral, but—

Well, that's great.

Yeah. But we— I can't guarantee it absorbs yet, but when we can, I'll let you know.

Well, let me know. And CMAX, of course, as you know, it helps with dopamine and dopaminergic, you know, that dopaminergic activity. Which is part of the Parkinson's things because those dopamine receptors and they're not expressing enough dopamine and their dopamine receptors aren't working correctly.

Well, CNET, that's the whole key, right?

Yeah. It's the key for, for Parkinson's.

So I like those two simultaneously together because I had also dopamine for depression, right? Motivation, all that. And Sribrelison, I think you were using?

Yes. And actually this guy did get cerebrolysin too, because I could get it back then. You know, it's now we can't get cerebrolysin. Okay.

Can you just, uh, explain what cerebrolysin is?

So cerebrolysin is actually a peptide that actually comes from pig brain, but it actually, uh, it actually helps with the neural connections. So there's some, some theories that it actually helps with some of the enzymatic functions in the brain. So the enzymes are working correctly, the neural connections are developing and working correctly. And there's some articles actually on helping with neurofibrillar tangles as well.

So, you know, there's, there's some good goes along with Alzheimer's and, and yet it increases connection. So in theory, it should make you smarter, right? You know, the more connections and yeah. So recently the FDA said it's a biologic. But it is— it will be available orally, which actually studies show it works through EEG studies showing it.

So, well, that's great.

But, uh, I love serpilicin as well. It's, uh, it's been around forever. Yeah, and used in Europe as a standard therapy for Alzheimer's, traumatic brain injury, you know, and anything neurologic. And we are so behind on all this stuff. And like, right, I'm sure when you give lectures, it's like people go, why haven't I heard of this stuff? Right? And they've been around forever because they've been around forever and there's no patent on them. That's why you don't hear about them here, you know?

Right, right. And Cerebrolysin was great. I used it on a couple of football players, ex-football players that actually had a lot of brain injury and brain trauma. We actually, you know, we had some MRIs actually showing a difference in the brain volume, well, before to after. And so, and I used to use it in some old things and then when now I don't want to use them because FDA doesn't want us to use it anymore. So I don't want them to think I'm using it.

It is. It's like, hey, if it's safe and it works, they're going to try to take it away.

Yeah.

And I remember I was at a stem cell conference and actually in Florida and it was NIH, I think. And they just studied, they could tell what position the football player played. Freshman football player by where the brain damage was.

Yes.

Now, and, but they could, you can reverse it.

Yeah. Yeah. And they're showing it, you know, not just even by cerebral lysin, but there's other things.

Yeah. Yeah.

I know there's so many things that are, are becoming tight for us to be able to even get.

And then you're beginning to worry all areas scared of, you know, all the stuff's gonna be taken away. I'm thinking like, okay, what country do you want to move to? Or why don't we all get together and buy a yacht, park it 4 miles off the coast. We can do whatever we want.

I thought about that same thing. I thought I could just, you know, get a yacht and go 4 miles off the coast. And then I thought, well, you know, it's a quick flight down there to Barbados. Maybe I could go to Barbados and, you know, Yeah, no, it's— I paid $250,000 to become a citizen of Barbados, so I can become a senator.

Senator.

Yeah.

All right. Maybe we get a 2-for-1.

Yeah.

I think I have a coupon.

There we go. So, you know, I also did some other things with him because I really believe just like in the Alzheimer patients, you need the proper fats for the brain. So I always get of them phosphatidylcholine. Oh, I love it. I love it. And so phosphatidylcholine is one of my things that I use a lot of IV-wise, and I use a lot of it orally. And it helps a lot of these patients because almost every one of my patients has some kind of neurological insult. So that is—

I like it better for chelation, you know, getting rid of heavy metals, toxins. Yeah, I think it's great.

And I think it does a great job and people tolerate being— going under chelation better with phosphatidylcholine than with EDTA or DSMA.

Oh, yeah.

So, you know, I think that they don't have to go under those harsh circumstances and we still can get some great results. And I've actually found that for some of the environmental toxins, like Roundup, you know, when I test somebody, you know, everybody has Roundup in them, right?

Is it still on the market?

Yeah. So, but, you know, that, you know, I test a lot of those environmental toxins, but I find that phosphatidylcholine really— I can get good results of bringing those down very nicely with phosphatidylcholine. Not any other agent, just phosphatidylcholine. So that's why I really love it, because I agree with you.

And the studies on BPC-157, you know, shows that it'll protect the body from numerous toxins. And even in studies on alcohol, you know, protect the liver from basically high liver enzymes, protect from overdose of medications, prevents a hangover too. Yeah, but mycotoxins, neurotoxins. So it's nice, it, you know, prevents that damage from them.

Yeah.

Yeah.

Yeah.

We're just, we are bombarded with toxins. So it's like everyone should be on it.

Really. Really. We all should be on BPC-157.

Yeah.

And if my kids on it, like again, what's the downside?

We can say cost. It's not that expensive. You can do 1,000 times the dose. They can't find a toxic level.

Right. That's the nice thing about it. Right. And that's the nice thing about a lot of these. You know, a lot of the peptides, nobody's ever been really— there's a few that, you know, has some toxic issues with them, but I haven't found hardly any of them to really have any problems at all.

You know, I've heard that to medication, any medication, you know, I mean, over-the-counter antibiotics. I mean, or when you look at the studies on like TB4 and BPC, you can take 1,000 times the dose. No toxicity. Try that with anything. Tylenol, try it with water.

Yeah.

You're gonna die. It's less toxic than water.

Yeah. Well, our water's pretty toxic. So he was ecstatic. So he actually, you know, began to be able to move quite normally, really take him off any of his other medications.

And how long after kind of starting this stuff?

So he started feeling more aware and more— his cognitively, he felt like he was back about 2 weeks after starting everything. So 2 weeks. And really, when you began to look at his inflammatory markers, his inflammatory markers came down. It took 3 months for his inflammatory markers to really come the whole way down to a normal range, but they came down. They were in the normal range, you know, his The last time I checked him, and M11 to the CRP was 11 or something. Yeah, 11.4. And he came down. Well, he was still, he was, let me look back what he was 3 months out. He was still up there a little bit, but he was 6.2. But you know what he is today? Because I just saw him not too long ago. So I'm pulling up his labs here.

Hey, we know what to say, actual patient.

I saw him, I saw him 3 weeks ago. This is 3 weeks ago and his CRP was 0.4.

You're the man. I hear the man.

I love it.

I love it.

So that's great because, you know, now it's a year and a half out and he has more of a life. He goes out with his wife. They walk every day. They do all kinds of things. They moved from New Jersey down to the Villages. So now he's actually both.

Think of the impact on his life.

Yeah. Why?

What did the neurologist say?

Well, the neurologist was saying, ah, this couldn't be. It's just, it's just chance. It's by chance. This just—

Oh yeah. So I would rather believe it's a miracle.

Yeah. So, and they said your medication's just now started working. So make sure you keep taking his metal.

That's good. Yes. Usually takes 4 years to kick in.

Yeah. Now I've had some patients that have gone back to their doctors with neurological issues and they've gone back in there, you know, and he said, oh, you're not using a cane to walk in here. And he said, I must have misdiagnosed you. And he walked out and never saw the patient again.

Oh, I've, yeah. Have you seen ALS patients at all?

Yes.

Yeah. So I always— patients come in, they're in a wheelchair, they're jogging, they go back to the neurologist. Oh, must be a misdiagnosis.

Yeah.

And they'll never admit it was the treatment. Or one patient with a hurtle cell cancer, and her doctor was like, you have to get this taken out tomorrow, you're going to die.

Yeah.

And then she came and saw me. I said, I think we can reverse it. I'm not saying what, you know, the, you know, but I said, give me 6 months. And we did. Got the scan, the biopsy. It's gone. She goes back to her doctor, who she was very friendly with and thought he'd be so excited. He wouldn't come out of the room and just said— told the front desk to tell her she's no longer a patient.

You know, I thought we were supposed to be happy for our people and our patients when they got better, no matter if I get them better or somebody else. Yeah, better. You know, I'm always excited when somebody gets better, you know, and I don't have to be the one that gets them better for me.

Just, hey, if I could— yeah, if you can learn something from someone else, awesome.

So we got to learn from each other. And I don't understand the hostility between all the physicians, you know, and why we're trying to cut each other's necks.

But let's see, anything else? I'm going to babble so long because I love hearing all your stuff. Um, we're going to go on forever. You want anything else with that one or I'll move to the next one?

Let's move to the next one so we can get some people with some gut issues.

All right. Sounds good.

So I have, because of my own history with Crohn's, so I had Crohn's as well. Oh, not just throw that in the mix. Not just Lyme and mold, but I had Crohn's as well. And Hashimoto's. Let me. But, uh, so, so I get a lot of Crohn's patients just because in, in one of my books I talk about the Crohn's issue.

So, oh yeah, just back to the, uh, Parkinson's. Did you check for Lyme or anything with these?

I did because of his C4A being high and C3A being high. So he had both of them high. So I checked, you know, all of his other, his ACTH, MSH, uh, ADH, they were all normal. He, his Western blot, but I don't go just totally on that. So, you know, So we, so we went to the IGeneX route and I did that test and he was negative on that. So then I did some PCR. I did the PCR test too. Um, so, and he was negative on all of that. So he did not have any Lyme, which I was kind of surprised because it's, yeah, it's like, has Lyme.

Infectious until proven otherwise.

But yeah, you know, you gotta he did have some environmental toxins, and I have to be honest, he did have some environmental toxins. They weren't the worst environmental toxins I've ever seen. You know, I see healthy people that have that amount of environmental toxins.

Yeah, but again, it goes along with, you know, genes and all the other stuff. Stress is huge. And what else? It's like if someone has Lyme, I think they're gonna be fine, but then you add on stress and mold and genetics that, you know, but yeah. So it's just we're being bombarded. And it seems to me like, I mean, at parties or whatever, it's like everyone coming home. I'm sick. My sister's sick. Like, you know. Oh, yeah. I'm sick.

Yes.

No, they know.

I went to a party the other night and I was like, oh my, everybody's coming up to me. Oh, I need help. I've got this or I've got that. And I'm like, oh, did this?

No. Like, well, you know, I should do this, this, this, or test this. No, my doctor says it's not it. And I'm like, okay, how's that working for you? You know, okay, but, uh, right, exactly.

So this, this, uh, was one of my favorite, favorite patients. She was a 42-year-old female. She had 3 children, kind of real, you know, she really wanted to be active. And she was on biologics, of course, like most Crohn's patients, they're all on some kind of biologic. But they were working for her.

Oh, yeah.

Yeah. And they were making her so, so fatigued. She says, I can't even enjoy playing with my children anymore because I'm so fatigued. Everything's out of whack with me. I just can't figure it out. I'm just tired all the time. And she actually— her father is a dentist around here, and her father had heard, had some patients that came to me. So that's how she ended up coming to me. And So she, she came in and really, you know, all her numbers were perfect. And I'm like, I've not seen this before. My patients that are just coming in for anti-aging, I'm all like, they don't even have this good a number.

Yeah. Like people say, what if you don't find anything? As it hasn't happened yet, you know?

Yeah. So I was just kind of shocked, you know, how good her numbers were, but you know, her gastroenterologist had shared all the slides and, you know, all the things. So she really did have Crohn's. Nothing was helping her with her Crohn's. And she was losing some weight and she did a lot of yoga and she already ate pretty pristine, although she did like— she was Italian and she loved her pizza and her pastas. So she ate a lot of gluten.

A lot.

And I kind of addressed that right away where let's stop the gluten. Let's try to get gluten-free things. Cause there's a lot out there that's available.

Everything is tough. Yeah.

But so, um, but it's hard for the Italians to do away with that. So she actually was in one of the new local newspapers around here because of her story, which actually, you know, was talking about peptides.

So, oh, I was going to say congratulations, but you never know.

Yeah. Well, I'm going like, oh my goodness. Now I have a target on my back. Right. So I was a little bit worried about that because she told her story, you know, in this newspaper. And I was like, oh no, I really don't want this. I don't want that kind of publicity.

No, it's, it's scary. You find this safe treatment that helped someone with a horrible illness.

And first you think of, oh no, you know, it's this, I don't know, the healthcare system in the world is Yeah, so, you know, really, I, I started on BPC-157 right away, but I also did some thymosin alpha 1 because I really think that that really helps with modulation of the immune system because, you know, Crohn's is really, it's really kind of an autoimmune process. So I always do that with my Crohn's patients or anybody with autoimmune. I'll put them on thymosin alpha 1. That's just what I do. It's— and so, you know, she was on both of those from the get-go, from the beginning, and she really started getting some benefit. But I don't know if you've read any of the articles on how sea lang actually helps with the healing of the gut as well. So I actually did put her on some sea lang, and that's when I had some sea lang that was— you didn't have to do it intranasally, you could actually give a sub-q injection of it. So we did sub-q injections for sea lang for her. And you know what? She came off all biologics because they weren't really helping her anyway.

Yeah. And you listen, you lame other immune modulator, you know, it's that immune system.

Yeah. So I think it was primarily her immune system that really was out of balance and really causing that issue.

What was her human transforming growth factor beta, C4A, natural killer cell function?

Her natural killer cells were great. They were normal. And, and so I was kind of surprised. I was really surprised, but she, she was good. Her TGF-beta was normal. C4a.

I would have bet money, you know, someone with Crohn's that, but it's interesting, just a little kind of similar story with an 8-year-old girl that mom brought in. They tried everything, going to do a colectomy, said, uh, you know, and, uh, basically, uh, EPC-157, the TB4 fragment orally, and just dramatic change. And most of some, some stem cells are really— what are they now? Signaling cells. And these are the cells which are what? The decellularized growth factors. Yeah, yeah. But dramatic improvement, right? So no longer neoclectomy. And then she went and had dinner with actually trying to be my partner and said, oh my God, we're so happy with this doctor that, you know, they're going to do surgery on her. And he got her better. And he's like, my partner's like, is it Holtorf? She goes, yeah, do you know him? It's like, yes, my partner. But so it was funny as a family friend of theirs. I think it's—

yeah, that's great.

An 8-year-old, same to colectomy, you know?

Yeah. Yeah, I have a, I have a little 11-year-old boy like that too right now. And he's He's so much better.

You know, I imagine you treat autism.

Yeah. Yeah.

So, but don't the autism kids look just like the Sears, the Lyme? They have.

Yeah. And they're so much the same. So I have a story about that because my 22-year-old son was on the spectrum. You know, he had the flapping hands. He couldn't look at you in the eye. He just really socially was very awkward. He's a very intelligent boy. But just socially was really, really awkward and really had a hard time. He went to go to college and he couldn't even go because he couldn't go into a classroom because he got so distraught and, you know, and it was just— so it didn't even work out. So I paid for college for nothing. But yeah, I started treating him and, you know, we got him a lot better. So, you know, there's peptides, those peptides work well for those Those, and I like Stan Stronghold and the exosomes and, and the peptides are great.

Yeah, I could tell you a bunch of stories of, you know, kids where the mom sent in a video playing basketball, the basketball just went by him. And then we have to do them before anyone comes in because just throwing F-bombs like crazy, no one get near him. Now he comes in and goes, can I have a hug? He's sarcastic. He gives them shots and they show them the gymnastics and actually playing basketball.

Yeah.

Pretty amazing. Yeah.

That's what, you know, and that's like my son. Most people would look at him now and they said, he's normal. He's normal. He's not the same kid he used to be. He is normal. And he actually is now working for 8 different doctors, you know, and he's their medical assistant and he loves it, likes to stay busy and you know, but he's out there, he's social. It's wonderful. So that's awesome.

That's great.

So she was doing great. Now, this is one story about her I want people to know. And I think it's really critical that people realize that they're going to have to pulse these things on and off the rest of their lives. They just got better. And then you walk out there and you're great. Because She did get decellularized growth factors as well. And, you know, she did so well from that. Her bowel movements were normal. Everything was completely normal. She loved it. She felt great. She had more energy. She probably about 8 months to 9 months later, she started feeling bad and she didn't come back in right away. She didn't say anything. She just thought, oh, it's going to pass. It's going to pass. Well, then, you know, before long she starts getting worse. And then she comes in and it's like starting all over from scratch where if you realize that this is going to be the thing, you know, if you have BPC-157, you can take it all the time. But if you don't or you can't afford to take it all the time, at least pulse it once you're better, you know, because you're going to have to be on that on and off forever.

For whatever reason, for, you know, the stress, genetics, the toxins they're exposed to, mold, whatever they got going on. Yeah.

Well, yeah, and that's the reason. And when she came back in the second time, she had Sears markers that time. So she really did, and her genetics really showed that she, she had HLA where she really couldn't get rid of the mycotoxins well. So her mycotoxins were probably, you know, we needed to bring them down and we needed to, you know, get those Sears.

What therapies do you like for getting rid of mycotoxins?

There's something I really like. It's called Mycobind and it's by PHP.

I like them. I like them.

Yeah. So that Mycobind, I see their results for their mycotoxins come down pretty dramatically. Not right away, but over time. And I love that because of that. And I think that combined with phosphatidylcholine tyropholine is kind of essential for bringing down those mycotoxins.

Yeah, well, with the Mycobind, like, I think, you know, like the cholestyramine, it's just so like you can't— it binds everything else and it's such a hassle. Is it easier to take the Mycobind?

Yes. So the Mycobind, I don't think it's binding all the nutrients like the cholestyramine. So cholestyramine, a lot of, a lot of that, my serious patients never tolerated it. Bad. No, so you put them on Wellcall or— I mean, they were all the same. Nobody tolerated them. So I had to find something else. So, you know, when this came available, I was so ecstatic because I was actually asking for it for a long time. Like there was a couple other doctors asking for it too for quite a while. So I love it because it has the okra and the beet together and it has that fiber component from both of them that is actually the proper electrical charge. So you have to have the right electron charge to actually pull those mycotoxins out, right? So people that are doing charcoals and stuff like that, they don't realize that the charcoal is actually the same charge as the mycotoxins. You're not actually pulling mycotoxins, you're pulling other toxins.

So I don't know, and this like, you know, kind of major, you're getting down to this you know, biochemical cellular level. And I'm just thinking, how many gastroenterologists talk about that?

You know, not too many. So, you know, you know, we did get her better again, but, you know, it did take a little bit longer the second time around. It wasn't— she wasn't better in 3 weeks.

Did you ever check her house for mold or anything?

Yeah, I had her check her house. So immediately when she came back in and I realized, hey, you've had a mold exposure. Well, then I found out the whole thing is she was separated from husband. She moved out, she moved into a place. So she had all this stress, a lot of stress, and then she had moved into a moldy apartment building. And I'm going like, this is what's going on. So you gotta get out of there. You have to be in a, in a safe place. Thank goodness you're renting this place. I always encourage everybody, if you're renting, have your house in have that place inspected before you move into it.

Yep. And I burned up when the— like, you know, and having some inspectors, it's, you know, hard to find, you know, are they going to— especially ones that do the remediation themselves, like, oh, there's mold everywhere, you know. And well, what tests do you like for home?

For the home? Well, I use a guy. His name is John Bodie. He's an environmental guy. He does a lot of the government buildings as well, and he tests them for mycotoxins. So I use him for testing, but if I'm gonna just test real, you know, some people use the ERMI. I don't think that's so accurate always. There is something from Realtime where you can actually swab the filters in your furnaces and you can actually send that off to see if you're having them floating out through the house. Because a lot of these molds are behind the walls and you don't see them.

Yeah, I want to know what's out in the air.

You know, that is— but the gases are coming through and it's really hard to test those gases. So everybody wants to test the mold that's out here in your room. You might not have mold. You're going to have the gases out here because the mold's probably behind the walls. Good point. Good point. So I want to know what the mycotoxins are in my house. And so I use the real-time, you know, I do that. I swab ours from time to time. And then I have a friend comes in and says, I want to open a can of worms, but EMF, thoughts on that? Do you? EMF? I put EMF blockers everywhere in my house.

Yeah. So I have a guy coming over tomorrow to our house and then kind of building a house, but, and to check everything, you know, and builder let everything get moldy. He's like, oh, don't worry about it. I'm like, no, I'm worried about it. I mean, you see black mold everywhere. So yeah, but that's a mold guy. But yeah, the EMFs were either, you know, and I told a bunch of people like, oh, just turn off your Wi-Fi at night. And people, I don't believe in that. But a couple people did it and they're like, I slept better. And then like, I've all of a sudden I didn't sleep and I realized I didn't do it. I didn't, you know, and started sleeping better. So I think it's another area. I just wait till 5G, you know?

Right, right. I'm a little concerned about that, but you know, our Wi-Fi turns off automatically every night in our house. In fact, that's why I came to my office to do this, because it's made out of steel.

And it makes mold grow faster too.

Yes, it does. So now I do EMF blockers, like on my laptop here, I have an EMF blocker on here. My, you know, I—

Well, what do you think those work? Like the little dots and things like that?

I'm not sure how well they work, but I feel better when I have them on. And I have one on my phone. I put them on my kids' phones. So I feel better when I have them on versus don't have them on. So I don't know how much of a thing that is, and I don't know how to actually study these things and test these things to see if they're really working or not. And so that's what I'd like to find the science on these things to see how—

I'll ask the guy tomorrow. He's like the man supposedly, and he's gonna come over and analyze our house and everything.

So, well, when you find that out, you let me know because I don't have an answer on that stuff yet. Because I'm gonna—

I didn't think to ask him that, but I'm going to ask him that.

I really haven't studied that enough, to be honest with you. I have some EMF patients, but most of them actually have mold.

It is, it goes together. Like they just get sick, you know, and where other people go, it's crazy because I'm fine and I'm in the, you know, and I'm exposed. It's like you have, you know, with mold people, they're in the same house. It's like one person or whatever is the one that's sick because they have the, all the other stuff going on.

Right.

And I think it's all, you know, and you were— vaccines, you know, who's going to get vaccination harm? You look at their immune system. If their immune system is off, they are much more likely. I think it, you know, it's kind of a marker and a cause, you know, to that whatever it is makes them much more prone. For instance, when I had Lyme, and if I had a flare, I'd be allergic to everything. And when I didn't, I wouldn't.

Yeah.

Yeah.

Yeah. It's kind of strange how that goes, you know? And I know when my mycotoxins levels are going up in my body, you know, and I live in Florida, so, you know, we're pretty humid out here. It's pretty humid. You know, I have to be kind of careful, but I like to go over to the beach, you know, kind of a lot. And you can tell because, you know, I I've got some sun on me because I go to the beach a lot, but I love the salt air. It just, it really cleanses those negative ions.

It's like, uh, or even in the shower, I show them to think better. That's why I have a little notepad that you stick because, you know, all your great ideas are like in the shower, like, and then, but then you forget when you get out.

Yeah.

Yeah. Same at the beach. You just kind of clears your mind and you think better, but again, I'll I'm babbling too much. So let's go on. I think we have last one.

So I have two we could talk about.

Either do—

oh, another Crohn's patient, or we could do this. I have a patient. He's interesting. He is a— why don't we do this one? He's a Tourette's. So a lot of people think, oh, Tourette's, you know, wow. Well, he's a 44-year-old. You know, he was a father of triplets. He was busy. He was always working. He was a hard worker. And so he didn't have Tourette's as a younger person. He really developed Tourette's around the age of 40. And it was just kind of suddenly. And so trying to figure out his history, it was really kind of difficult. But he started having some random jerks. So then he, you know, he was a carpenter with his dad and he couldn't really do the carpenter work anymore. And so he just started getting worse and worse and worse. And to the point where, you know, he's making the grunts and the noises and he can't control anything, but he's also getting self-consciousness and abuser. So, and then he started getting muscle rigidity. So he did, he was from Alabama and he went to the, one of the major medical institutions there. And, you know, they put him on medications, but the medications did nothing for him.

He was Antiseizure meds and stuff, slow everything down. Yeah.

So he was on a lot of antiseizure medications, but he was also on lots of high doses of benzos. And I'm like, my goodness, you know, he was just, he was really on a lot. So, uh, when I did his, uh, results, he actually had positive for SIRS. He was a great— he was really a SIRS patient. And I really thought, did this SIRS really induce a Tourette's type thing? I've seen SIRS, you know, for people with dementia, people with ALS, people with all kinds of different things. But, you know, that was my actually first Tourette's patient that actually was positive for SIRS. And so I thought he's—

can you kind of define SIRS for everyone?

Because yeah, so SIRS is chronic inflammatory response syndrome. And chronic inflammatory response syndrome usually happens because of biotoxins, toxins put off by biological organisms like mold, mycotoxins. But they can also— you can have SIRS because of toxins put off by Lyme and by other— Babesia, Bartonella, you know, you name it, you can have those toxins. All those things are going to put off some type of toxin. So he actually had had a mold exposure, not realizing it.

And my thought is that the— I think the combination of stress, a chronic infection, and then a toxin. So I think there's often an underlying infection.

Well, he had high Epstein-Barr titers too, which—

and though Lyme—

his Lyme was negative, but he had high Epstein-Barr. And I was surprised. He's from Alabama. I was surprised he was negative. He was one of my first Alabama patients that was negative for Lyme.

But there's no Lyme in Alabama.

Yeah, right. There's no Lyme in Florida either. Yeah. So for him, you know, his TGIF was 32,000.

Wow.

2009 TBIM.

Tons of inflammation. His, his brain was just inflamed.

Yeah, his brain was inflamed. So Vibrant of America has something called a NeuroZoomer.

Oh, so I was going to ask you that because yeah, that's, I didn't recognize the name of the company. Yeah, NeuroZoomer. How do you like that compared to the other, uh, PANS test?

Um, well, I, I use the PANS test too. Too. So I can't say that I use one or the other more, but I do the PANS test too because I belong to that society.

You want to take on everything, man.

So I like them both, but this one just seems that it hits a few more. So he had some antibodies to some things that really were key and instrumental for the anticholinergic things. You know, he had he had antibodies for the blood-brain barrier. He had a lot of inflammation in the brain, basically. Yeah. So, and, and so because of his inflammation in the brain, I said, we've got to pull your inflammation down. It's got to be number one. So, and naturally for the brain, you're going to need fats, right? So, you know, one of the first things I do is start IV phosphatidylcholine on him and, uh, But I also started doing— I did something. I said, you know what, I want you to get this fast. So I want you to know this is kind of my first trial of doing this. So my first trial of doing it, as I filtered out thymosin alpha 1 and I put it through a filter, and I actually gave him thymosin alpha 1 through IV.

Okay.

But I filtered it.

Yeah, no, hey, we, we due to peptides and some studies that it's in on thymosin beta-4 and TB4 frag is actually a much smaller cross the blood-brain barrier, better barrier, better and suppressing microglial activation.

Yeah, the microglial activation is key in a serious brain on fire. Yeah. Yeah. And that's basically what I had was a brain on fire. I started giving him the thymosin alpha-1. He was on BPC-157 because he had gut inflammation too. And he, of course, BPC-157, I love for everybody anyway. So I'm just making— this works for everybody. So he was on that. We did do TB4 on him as well. And, uh, I did do decellularized growth factors on him, which are great. They're great anti-inflammatory. They do, they do cross the blood-brain barrier pretty well. And really brought a result for him, you know, but he, he does come down every 3 months and just do a little bit of a tune-up with me.

Did he stop having Tourette's?

Yes. Yes.

You should write that in a journal. I mean, you should, you know, right?

Yeah. I'm in the process of writing his—

so there are any cases of that? I'm like, you know.

I don't know, he's diagnosed with Tourette's. But you know, I'm like, did he truly have Tourette's? I don't know. Was it really more of the Sears? He had zero information.

Oh, he didn't really have it. He just likes to scream and cuss at people.

Right, exactly. You know, I mean, his, his kids really loved it. And he went back to doing his artwork too. He was an artist. He loved to go back to doing his artwork. So it did take me a while a long while to get his TGIF beta down and his natural killer cells up. He didn't have any natural killer cells.

And so, you used melanotan, I think you said?

Yes, I used melanotan on him as well.

Can you talk about melanocortin?

Well, I love that because it's melanocortin. And as you know, that helps with neurological. So, and it helps what a lot of people think is just basically for the ghrelin system. But, you know, there's evidence that it really helps with serotonin, serotonin uptake, and it helps with neuromodulation. So that's why I like that melatonin system, because it's so key in the circuitry of the neurons. So the neurons really respond to the melatonin system.

Yeah. And again, we're— KPV, which is a fragment of that, coming out what's more potent and very anti-inflammatory. But yeah, the Lantania, known as the Barbie doll, because you get weight loss, increased libido, and you get tan.

Yeah.

I found it's great for younger people, but if you're older, then you get like dark spots and—

Right.

And I actually tried it. I'm like, this isn't where I'm very ADD. This is working into more and more. All of a sudden, I am just so dark. I'm like, some strange— like, I don't know from what continent or whatever, but I'm like, oh my God, you know.

Yeah.

But, uh, and that's kind of a problem. He was like PT141, um, erectile dysfunction, that, you know, it's another melanocortin which, uh, can cause some skin darkening, which is A little bit's good, but in older people, like, you know, dark spots and stuff.

Yeah, especially if you're Irish, you don't wanna—

yeah, yeah. So the KPV, again, is just the tripeptide, uh, has all the anti-inflammatory but doesn't have the melanocyte stimulating, so you don't get the skin pigmentation.

Yes, that's a great product, great product. And if it crosses the blood-brain barrier, much better too because it's a smaller fragment. Yeah.

Yeah.

And for mast cell, which is such a big problem, you know, mast cells are horrible. You know, we have so many people with mast cell issues today. You know, it's just become a huge, huge issue. And I don't see too many people addressing the mast cell issue, you know, yet.

But it's all, it's all the nerds on the Mast Cell Mastermind. Yeah. But they're still going after the direct. And I'm like, look upstream, you know, look upstream and modulate the immune system and you're gonna, you know, fix it. And also, uh, corticotropin-releasing hormone, yes, a huge mast cell stimulator. So, you know, if they have high ACTH, you want to give a little bit of cortisol, not a ton, not, you know, yeah, not prednisone or anything, but just bring that ACTH down, which will lower that CRH. But Right. Yeah, it's mast cells are huge. And even my girlfriend ended up like all of a sudden got dermatographia.

Oh, really?

And it's like she didn't feel too bad. I'm just like, wait a minute, what's going on? So that's when we called the mold guy in and yeah, we can't find any mold. And so, but of course she won't listen to me, whatever, you know, do this, you know. Oh, to send her to one of the other doctors in my office. Because God forbid she listens to me. But it's, it's crazy, you know?

Right, right. Yeah. And to matter of fact, you know, that's a mast cell issue right away. So she has way too much.

I, I do love that when you have a new patient in there and all of a sudden, let me just do and just take a pen and yeah, the flares up and they're like, oh my God, what? It's so common, yeah.

It's very common anymore. And so, that's why you have to— you really have to address that melanocortin system. But the melanocortin system really helps with a lot of these issues, with neurological issues, of course, because it does happen neurologically. So, that's why I like the central nervous system and the hypothalamic issues with that are kind of key.

That's like everything.

Yes.

And it's like the, like, inflammation of hypothalamus, you get diabetes, cause all the insulin resistance, and, you know, and of the brain, you get depression and anxiety and sleep disorders and pain, all that.

Exactly. Exactly. So, and that's what— and, and you know what, you, you address that, you address the hypothalamus, You know, these people with the pain syndromes, it really helps them a lot. A lot. Yeah.

And I did a review on HB axis dysfunction and chronic fatigue syndrome, and they found their, you know, I think chronic fatigue syndrome and fibromyalgia do a disservice to people because, right, like, oh, press on, you know, 11, 18 tender points, which they have nothing special about those tender points. Yeah, you know, the rheumatologist took like 20 minutes to do it. It's like, you got muscle pain, okay, you have sleep disorder, yeah, you got fatigue, okay, you got fibromyalgia. And then compared to chronic fatigue syndrome, which is symptom-based, you know, right? And they found the difference though was that the fibromyalgia patients had inflammation in the hypothalamus while the chronic fatigue syndrome more in the pituitary. And so which makes sense, pain center and the hypothalamus and Right. Yeah. So you fix that. And also, it was like 57%— no, more than that, 60-something percent of fibromyalgia patients had, you know, autoimmunity.

Yes.

You know, which just shows that this— it's all immune, all inflammation.

Right. And we're— and they develop autoimmunity in their brain. And so neurological autoimmunity is a real thing that we've got to start addressing. And getting it calmed down because that's why a lot of these people are presenting with neurological issues. And nobody can really pinpoint it because it really doesn't fit any specific category.

And doctors roll their eyes as soon as they come in and say, "I have fibromyalgia or Lyme." The doctors go, "Oh my God," you know? And I know you probably have this. It's like we have patients crying the first visit. When we do labs and show them on paper, You're right. Oh my God, don't believe me. And, you know, do you really believe me? I'm like, yes, it's right here.

You know, this is it. I see it right here. So that's what I love about doing what we do, because, you know, you get some— it's hard because you have, you know, they're demanding, the patients are demanding. They've been sick forever and they've been neglected and they've been told they're crazy and it's all in their head. I know I was told that.

I have tried so many things where doctors say it's placebo. But they don't respond to placebo because nothing's worked.

Right, right. And so, you know, when you, when you help them, you know, it's just worth it because it's just, it just makes it so worthwhile.

Yeah, exactly. That's, that's why you can— it's tough, but go in again the next day.

Yeah, yeah, exactly.

It's awesome. I just, I could see you're just changing so many lives on a daily basis that you're probably like, people are so thankful. Like, oh yeah, yeah, that's what I do.

Exactly. I just have to— we have to just train enough other physicians because I can't treat everybody.

It's true. Are you just by yourself?

Yes, I'm just by myself. You know, I, you know, I kind of would like a PA or nurse practitioner to come in with me at some point here. So we're just recovering a little bit from COVID too. So once we recover a little bit more—

I love seeing my assistant.

Running around. So yeah.

So awesome. This has been great. I don't know how long we've been talking, but I just don't know what time it is. I just love all your information. And you're so humble too.

Oh, thank you. You are too. I love your knowledge base too. You know, it's just like, so great. We're gonna have to get together.

Hey, I've been thinking of some things actually while we're talking. But again, because we're certainly like-minded and your passion is just amazing.

Oh, thank you. So is yours. I'm glad you're getting the fragments of these things and getting them so that we can actually take them orally and really getting, you know, where we really need to get with these because, you know, the FDA is not going to come against a fragment of something. Very well, that is, we're trying.

I didn't know how much work it'd be and how expensive, but, uh, yeah, I wouldn't have done it if I knew. But, um, yeah, I love it, you know, just like you. It's like researching, which could take so long, but doing funny little nuggets I don't know. It just makes it. And lose a lot of sleep. I don't have much balance in my life, but, uh, yeah, I think my tooth just came out that I knocked out in my accident. I broke my nose. But, uh, anyways, so yeah, go back to this from Amazon and, and glued it in.

Yeah, how do you get the dentist broken and the guy screwed it up?

So look, I'm mad. I'm not mad.

So, but you still look good.

I have a— from LA, but awesome. Hey, thank you so much. Yeah, fuck you forever. And thank you for taking the time. I mean, I know so many people pulling out your coat and wanting your attention. So thank you.

You're welcome. You take care.

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