Dr. Ron Najafi explains how fructose damages mitochondria and blocks ATP production, forcing cells to convert excess calories into fat instead of energy. The liver packages this as triglycerides and sends it to fat tissue, a pathway he says drives obesity even in newborns exposed prenatally.
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It's a mitochondrial toxin. It inhibits mitochondrial ATP generation. So you have all of these calories and no place for them to go, and so they generate fat. So your cells are starved, but you're generating fat instead. And that fat has to go somewhere. When the fat is in the liver, the liver has to get rid of the fat. So it packages it into a molecule which we call triglyceride, goes into the bloodstream, and then insulin will offload that fat into adipose tissue, into fat cells, and that will grow your fat cells. So the combination of fructose induced triglyceride plus insulin equals adiposity. So that's one of the drivers of obesity. It's not the only driver, but it's a big one. And it's one that kids are exposed to, and it's one that even fetuses are exposed to. And it's our best assessment as to why we now have an epidemic of neonatal obesity.