Robert Lustig:How Aspirin’s Impact on ACAT-L Makes It Mitochondrial Toxin Linked to Reye’s Syndrome
Robert Lustig (speaking in a YouTube video uploaded by Dr. Ron Najafi) asserts that aspirin — specifically its metabolite salicylic acid — is a mitochondrial toxin that causes hepatic mitochondrial dysfunction, altered hepatic lipids and triglycerides, and induces a defect in the long-chain acyl-CoA dehydrogenase enzyme (ACADL). He links this mechanism to Reye syndrome in children who received aspirin during a viral illness, arguing the aspirin-induced mitochondrial shutdown caused the fatalities.
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